Tuesday May 22, 2018
Home India Modi Govt. to...

Modi Govt. to set up cancer centres in coal mining areas

0
//
27
credits: indiatimes.com
Republish
Reprint

Piyush_Goyal_January_2015Kolkata: With the central government setting up cancer centres in coal mining areas, union Coal and Power Minister, Piyush Goyal on Friday, sought assistance of the Tata Medical Center (TMC) in establishing the medical facilities.

“I invite and request Mammen Chandy (TMC Kolkata director) and other colleagues, trustees of the hospital to consider helping us in setting up these centres. I invite Chandy to partner with Coal India Limited to help set up these centres,” Goyal said here.

The minister inaugurated ‘Premashraya’ — a residential facility for underprivileged cancer patients at TMC here.

Goyal said the centres will come up in Jharkhand, Chhattishgarh, Odisha, Madhya Pradesh, West Bengal, and Maharashtra and will be subsequently extended to the western parts of the country.

“Certainly, we are not doing any help to the people living in the mining areas… we can at least make sure we don’t allow them to suffer the consequences of large scale mining in these areas,” he said.

The minister also proposed the use of mobile mammography units for breast cancer screening in the mining areas. (IANS)

Click here for reuse options!
Copyright 2015 NewsGram

Next Story

New Therapy for Drug-Resistant Skin Cancer Suggested by Researchers

A team of researchers has managed to exploit a vulnerability in melanoma or skin cancer that develops resistance to a targeted therapy, providing a potential new therapeutic strategy to selectively kill the drug-resistant cancer cells.

0
//
23
The reason for increased bleeding is not known. It may be because rivaroxaban is more 'potent', the paper published in the Journal of Clinical Oncology said. (IANS)
Representational image, pixabay

A team of researchers has managed to exploit a vulnerability in melanoma or skin cancer that develops resistance to a targeted therapy, providing a potential new therapeutic strategy to selectively kill the drug-resistant cancer cells.

The study has shown that when cancer cells develop drug resistance, they also acquire a new vulnerability, the Xinhua reported.

The researchers, led by Rene Bernards of the Netherlands Cancer Institute and Oncode Institute in Denmark, exposed this new vulnerability in melanoma that has developed resistance to treatment with a BRAF inhibitor — a targeted therapy that blocks a signalling pathway in the cancer cell through which it gets the message to keep on dividing.

Since more than half of all melanoma patients have a mutation in this BRAF gene, the BRAF-inhibitor stops tumour growth in those patients.

But within a few months, the tumour cell adapts the original signalling pathway and becomes active again, and even hyperactive.

The researchers, however, found that the hyperactive resistant melanoma cells produced large amounts of reactive oxygen species, but cancer cells still sensitive to the drug did not do so.

Combining the new compound with vitamin D allowed certain protective genes to be expressed at much higher levels than they are in diseased cells.
Representational image, pixabay

The study, published in the journal Cell, found that the abundance of free radicals caused the resistant melanoma cells to stop dividing, but they did not die.

When tested on mice along with an existing drug, vorinostat, which is known to stimulate the production of free oxygen radicals, the researchers saw tumours shrink under the influence of the drug, the report said.

This laid the foundation for a new therapeutic strategy: Treating patients with BRAF-mutated melanoma, as usual, with signal pathway inhibitors.

When the tumour becomes resistant, stop giving those inhibitors and immediately treat the patients with vorinostat to kill the resistant cancer cells.

Also Read: Leukemia Progression in Kids Can be Delayed Through Bone Density Treatment

“It is not a combination drug. It is very important that you first stop the signalling pathway inhibitors because they suppress the free radicals and thus eliminate the effects of vorinostat,” Bernards said. (IANS)

Next Story