Friday January 17, 2020

Drinking Soft Drinks Linked to Obesity and Tooth Wear among Adults

Significantly, they also found that the increased consumption of sugary soft drinks may be a leading cause of the erosion of tooth enamel and dentine

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Soft Drinks, Obesity, Tooth Wear
The study published in the journal Clinical Oral Investigations, found that being overweight or obese was undoubtedly associated with having tooth wear. Pixabay

Researchers have found that sugar-sweetened acidic drinks, such as soft drinks, is the common factor between obesity and tooth wear among adults.

“It is the acidic nature of some drinks such as carbonated drinks and acidic fruit juices that leads to tooth wear,” said study lead author Saoirse O’Toole from King’s College London.

The study published in the journal Clinical Oral Investigations, found that being overweight or obese was undoubtedly associated with having tooth wear.

Significantly, they also found that the increased consumption of sugary soft drinks may be a leading cause of the erosion of tooth enamel and dentine in obese patients.

Soft Drinks, Obesity, Tooth Wear
It is the acidic nature of some drinks such as carbonated drinks and acidic fruit juices that leads to tooth wear. Pixabay

Previous research from King’s has found that tooth wear affects up to 30 per cent of European adults.

It is the premature wearing of teeth due to the softening of the dental enamel from dietary or gastric acids, combined with wear and tear.

Drawing on data from the National Health and Nutrition Examination Survey 2003-2004, the researchers analysed a representative sample of survey participants of 3,541 patients in the United States.

Patient BMI and the level of tooth wear were the exposure and outcome measurements in the analysis.

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The intake of sugar-sweetened acidic drinks was recorded through two non-consecutive 24-hour recall interviews where the patients were asked to provide details of diet intake across these two days.

According to the study, tooth wear is ranked as the third most important dental condition, after cavities and gum disease and the consumption of acidic food and drink is a leading cause of this.

Obese patients also have other risk factors such as increased likelihood of gastric reflux disease (heartburn) which was controlled for in this study.

“This is an important message for obese patients who are consuming calories through acidic sugar sweetened drinks. These drinks may be doing damage to their body and their teeth,” O’Toole said. (IANS)

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Researchers Find a New Mechanism to Prevent Obesity

New mechanism may safely prevent, reverse obesity

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Obesity
Stopping the obesity epidemic could be a critical aid in preventing and treating numerous cancers. Pixabay

Obesity, a global epidemic, is a known contributor to several cancers, including breast, colon, and pancreatic and in major breakthrough, researchers have discovered that a receptor found in almost all cells, called AHR, and known primarily to combat exposures to environmental chemicals, also plays a big role in the body’s metabolism.

Stopping the obesity epidemic could be a critical aid in preventing and treating numerous cancers and study, published in the journal International Journal of Obesity have found a critical target in this cause.

“We carried out experiments showing that when a drug named NF and known to block the AHR, was added to a high-fat diet, mice did not become any fatter than mice on a low-fat control diet,” said study researcher Craig Tomlinson from Dartmouth-Hitchcock Norris Cotton Cancer Centre in the US.

“Mice on the high-fat diet with no NF became very obese within the same time span. No ill effects were observed from the drug,” Tomlinson added. According to the study, the research team then asked whether blocking the AHR with NF could not only prevent obesity but reverse it.

Obesity
Obesity is a known contributor to several cancers, including breast, colon, and pancreatic. Pixabay

“In these experiments, we allowed the mice to become obese on a high-fat diet, and then half the mice were switched to the high-fat diet containing the AHR blocker NF,” Tomlinson said.

Over the next few weeks, the mice switched to the high-fat diet containing NF dropped to the same body weight as those mice on the low-fat diet.

The remaining mice on the high-fat diet became obese. No ill effects were observed, the researchers said. The research team investigated the mechanisms behind how the AHR, when blocked by NF, prevented and reversed obesity.

Using previous knowledge that the AHR regulates key genes in fat metabolism, the team discovered that in liver cells and in fat cells, the AHR, when blocked by NF, fails to induce several key genes required for fat storage and synthesis.

They concluded that the prevention and reversal of obesity from blocking the activity of the AHR is due to key genes regulated by the AHR that are involved in fat metabolism.

“Few to no studies have shown that obesity can be reversed by a drug treatment; it is even rarer to know the underlying cellular mechanism,” Tomlinson noted.

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The research team has begun investigating several key questions, including those around the dietary compounds in the food we eat that activate the AHR to cause weight gain , and the role that gut bacterial play regarding the AHR. Most importantly, they have initiated a clinical trial to determine whether the AHR may serve as a therapeutic target to reduce obesity in humans. (IANS)