Tuesday July 23, 2019

High Exposure to Radio Frequency Radiation Increase Risk of Cancer

Interestingly, the team found that rats exposed to whole body RFR lived longer than rats unexposed to any radiation

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cancer
Key gene behind breast cancer identified. Pixabay

Exposure to high levels of radio frequency radiation (RFR) — used in 2G and 3G cell phones — can increase the risk of cancer tumours in the heart, brain and adrenal gland, researchers have warned.

The study, led by the US National Institutes of Health’s National Toxicology Programme (NTP), looked at the effects of exposing rodents to extremely high levels of radiofrequency throughout the entire body.

While high levels of RFR caused cancerous tumours in the heart (found very rarely in humans), brain and adrenal gland, of male rats, the findings on female rats were ambiguous.

“The exposures used in the studies cannot be compared directly to the exposure that humans experience when using a cell phone. In our studies, rats and mice received radio frequency radiation across their whole bodies,” John Bucher, researcher from the NTP, said in a statement.

“By contrast, people are mostly exposed in specific local tissues close to where they hold the phone,” Bucher added.

For the study, the team housed the animals in chambers specifically designed for the study.

Exposure to RFR began in the womb for rats and at 5 to 6 weeks old for mice, and continued for up to two years, or most of their natural lifetime.

Breast Cancer
Cancer ribbon. Pixabay

However, the RFR exposure was intermittent — 10 minutes on and 10 minutes off — totalling about nine hours each day.

In addition, the RFR levels ranged from 1.5-6 watts per kilogram in rats, and 2.5-10 watts per kilogram in mice.

“We believe that the link between radio frequency radiation and tumours in male rats is real,” Bucher noted.

Interestingly, the team found that rats exposed to whole body RFR lived longer than rats unexposed to any radiation.

“This may be explained by an observed decrease in chronic kidney problems that are often the cause of death in older rats,” the researchers noted.

Also Read- Actress Richa Chadha Speaks Upon Patriarchal Society

According to the US Food and Drug Administration (FDA), while animal studies contribute to discussions on the topic, “this study was not designed to test the safety of cell phone use in humans, so we cannot draw conclusions about the risks of cell phone use from it.”

Since the exposure levels and durations in the studies were greater than what people experience, “we agree that these findings should not be applied to human cell phone usage”, the FDA said on Thursday. (IANS)

Next Story

Researchers Find Way to Make Cancer Cells Self-destruct

It also shows that ATF4 turns on the genes MYC needs for growth and also controls the rate at which cells make specific proteins called 4E-BP

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Cancer, Patients, Invasive
Traditional treatments often include chemotherapy or radiation to kill cancer cells and shrink tumors. Pixabay

In a new hope for cancer patients, researchers have found a way to cause some cancer cells to self-destruct.

The research team has identified a new pathway that works as a partner to a gene called MYC which controls normal cell growth, but when it is mutated or amplified in cancer, it sets off a chain reaction that helps tumours grow uncontrollably.

The pathway involves a protein called ATF4, and when it’s blocked, it can cause cancer cells to produce too much protein and die.

Published in the journal Nature Cell Biology, the study done on mice points the way towards a new therapeutic approach as inhibitors that can block synthesis of ATF4 already exist.

Cancer
Cancer Ribbon. Pixabay

“What we’ve learned is that we need to go further downstream to block tumour growth in a way that cancer cells can’t easily escape, and our study identifies the target to do just that,” said Constantinos Koumenis, Professor at the University of California.

According to researchers, this finding shows the alternative approach is to target ATF4 itself, since it’s the point where both signal pathways converge, meaning there’s less redundancy built in to allow cancer to survive.

Also Read: Consuming this Bacteria May Cut Risk of Heart Diseases

It also shows that ATF4 turns on the genes MYC needs for growth and also controls the rate at which cells make specific proteins called 4E-BP.

This study also found that when tumours in humans are driven by MYC, ATF4 and its protein partner 4E-BP are also overly expressed, which is further evidence that these findings may point to an approach that could work for humans. (IANS)