If you are finding it hard to deal with the pressure at the workplace, there is more reason to worry. New research has found that work stress and impaired sleep are linked to a threefold higher risk of cardiovascular death in employees with hypertension.
“Sleep should be a time for recreation, unwinding, and restoring energy levels. If you have stress at work, sleep helps you recover,” said study author Karl-Heinz Ladwig, Professor at Technical University of Munich, Germany.
“Unfortunately poor sleep and job stress often go hand in hand, and when combined with hypertension the effect is even more toxic,” Ladwig said.
The study included around 2,000 hypertensive workers aged 25-65, without cardiovascular disease or diabetes.
Compared to those with no work stress and good sleep, people with both risk factors had a three times greater likelihood of death from cardiovascular disease, showed the findings published in the European Journal of Preventive Cardiology.
People with work stress alone had a 1.6-fold higher risk while those with only poor sleep had a 1.8-times higher risk, the study said.
In the study, work stress was defined as jobs with high demand and low control — for example when an employer wants results but denies authority to make decisions.
Heart problems are a common development for people with diabetes and now researchers have found that diabetes is an independent risk factor for the development of heart failure in the community dwelling population.
According to health expert in India, if poorly controlled, diabetes leads to cardiomyopathy resulting in progressive deterioration of pumping capacity of heart.
“Diabetes is also a major risk factor for atherosclerosis and this eventually leads to blockage of coronary arteries. This leads to heart attack or myocardial infarction,” Satish Koul, HOD and Director Internal Medicine, Narayana Superspeciality Hospital, Gurugram, told IANS.
“Due to myocardial infarction, the heart muscle becomes weak and eventually heart fails as a pump leading to congestive heart failure,” Koul added.
According to the current study, published in the journal Mayo Clinic Proceedings, researchers evaluated the long-term impact of diabetes on the development of heart failure, both with preserved ejection fraction – a measurement of the percentage of blood leaving the heart with each contraction – and reduced ejection fraction.
They also looked at mortality in a community population, controlling for hypertension, coronary artery disease and diastolic function.
From an initial group of 2,042 residents of Olmsted County in US, 116 study participants with diabetes were matched 1:2 for age, hypertension, sex, coronary artery disease and diastolic dysfunction to 232 participants without diabetes.
Over the 10-year follow-up period, 21 per cent of participants with diabetes developed heart failure, independent of other causes. In comparison, only 12 per cent of patients without diabetes developed heart failure. Cardiac death, heart attack and stroke were not statistically different in the study between the two groups.
The study shows that diabetes is an independent risk factor for the development of heart failure in the community dwelling population. Furthermore, the outcome data support the concept of a diabetic cardiomyopathy.
This research extends previous findings and demonstrates that even without a known cardiac structural abnormality and with a normal ejection fraction, diabetic patients are still at increased risk of developing heart failure as compared to their nondiabetic counterparts.
“The key takeaway is that diabetes mellitus alone is an independent risk factor for the development of heart failure,” said study senior author Horng Chen from Mayo Clinic in the US.
“Our hope is that this study provides a strong foundation for further investigations into diabetes and heart failure. There is still much to learn and study in terms of this association and how to best diagnose and treat this condition,” Chen added. (IANS)