Thursday February 21, 2019

Obesity Spikes up Asthma Risk in Children

"Addressing childhood obesity should be a priority to help improve the quality of life of children and help reduce paediatric asthma," Finkel noted

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Obesity increases asthma risk in children: Study. Pixabay

Parents, please take note. Obese children are at an increased risk of asthma, a new study has found.

The findings suggest that the incidence of an asthma diagnosis among children with obesity was significantly higher than in those in a normal weight range and that 23 to 27 per cent of new asthma cases were directly attributable to obesity.

“Paediatric asthma is among the most prevalent childhood conditions and comes at a high cost to patients, families and the greater health system,” said co-author Terri Finkel from Nemours Children’s Hospital in Orlando.

“There are few preventable risk factors to reduce the incidence of asthma, but our data show that reducing the onset of childhood obesity could significantly lower the public health burden of asthma,” Finkel added.

For the study published in the journal Paediatrics, the research team analysed medical records of more than 500,000 children.

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The study provides new insight that could help us predict and manage diseases like asthma – which are a significant public health burden. IANS

The researchers reviewed de-identified data of patients aged two to 17 without a history of asthma, receiving care from six paediatric academic medical centres between 2009 and 2015.

Overweight or obese patients were matched with normal weight patients of the same age, gender, race, ethnicity, insurance type and location of care.

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The researchers found that obesity among children with asthma appears to increase disease severity. Being overweight was identified as a modest risk factor for asthma, and the association was diminished when the most stringent definition of asthma was used.

“Addressing childhood obesity should be a priority to help improve the quality of life of children and help reduce paediatric asthma,” Finkel noted. (IANS)

Next Story

Researchers Identify Genes Causing Obesity

Performing functional studies across other organisms, the team also identified two genes that were associated with significant increase in triglyceride and body fat across species

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India with 14.4 million had the second highest number of obese children in 2015. Pixabay

Researchers have identified genetic variants associated with obesity that is central to developing targeted interventions to reduce the risk of chronic illnesses like hypertension, Type-2 diabetes and heart disease.

The team from the University of North Carolina at Chapel Hill found genetic sites that affect human body’s size and shape, including height and obesity. The findings will help understand how genes can predispose certain individuals to obesity.

In the study, published in the journal Nature Genetics, researchers found 24 coding loci (or positions) — 15 common and nine rare — along chromosomes of individuals that predispose to higher waist-to-hip ratio.

Higher values of waist-to-hip ratio are associated with more incidence of diseases associated with obesity.

Obesity can now be cured by our body's natural weighing scales.
Obesity can now be cured by our body’s natural weighing scales.

“For the first time, we were able to examine, on a large scale, how low-frequency and rare variants influence body fat distribution,” said North.

“A better understanding of the genetic underpinnings of body fat distribution may lead to better treatments for obesity and other downstream diseases obesity also impacts, for example Type-2 diabetes and heart disease,” suggested North.

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Further analysis revealed pathways and gene sets that influenced not only metabolism but also regulation of body fat tissue, bone growth and adiponectin, a hormone that controls glucose levels and breaks down fat.

Performing functional studies across other organisms, the team also identified two genes that were associated with significant increase in triglyceride and body fat across species. (IANS)