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Your Genes Determine How Successful Your Married Life Is

They are relevant to how partners provide and receive support from each other.

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They are relevant to how partners provide and receive support from each other. Pixabay

Do you think you could lead a happy married life? The answer is in your genes, a new study has said.

Although prior research has hinted that marital quality is, at least partially, impacted by genetic factors, and that oxytocin may be relevant to social support, according to recent studies, variation on specific genes related to oxytocin functioning impact overall marital quality, in part.

They are relevant to how partners provide and receive support from each other.

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Husbands with a particular genotype were less satisfied with the support they were provided from their wives which suggested that it was also associated with being less satisfied with their marriage, noted Mattson. Pixabay

The study evaluated whether different genotypes – possible genetic combinations of the oxytocin receptor gene (OXTR)- influenced how spouses support one another, which is a key determinant of overall marital quality.

OXTR was targeted because it is related to the regulation and release of oxytocin.

“Genes matter when it comes to the quality of marriage, because genes are relevant to who we are as individuals, and characteristics of the individual can impact the marriage,” said Richard Mattson, Associate Professor from the Binghamton University in the US.

For the study, the team included nearly 100 couples.

Each partner was asked individually to come up with an issue to discuss something they identify as their most salient personal problem that was not related to their partner or partner’s family such as problems at work.

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Genes matter when it comes to the quality of marriage, because genes are relevant to who we are as individuals, and characteristics of the individual can impact the marriage,” said Richard Mattson, Associate Professor from the Binghamton University in the US. Pixabay

“We found that variation at two particular locations on OXTR impacted the observed behaviours of both husbands and wives, and that differences in behaviour across couples had small but cumulative effects on overall evaluations of support, and thus marital quality in general,” added Mattson, published in the Journal of Family Psychology.

However, what emerged as most relevant to overall marital quality for both partners was genotypic variation among husbands at a specific location on OXTR.

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Husbands with a particular genotype were less satisfied with the support they were provided from their wives which suggested that it was also associated with being less satisfied with their marriage, noted Mattson.

The researchers hope their findings provide the foundation for replication and additional study of OXTR as an enduring determinant of marital functioning. (IANS)

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Genetic Alteration Can Increase Risk of Developing Autism and Tourette’s Syndrome

Some researchers also found that the ability of the Thalamic brain regions to communicate with other brain areas was impaired by the genetic deletion

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Genetic deletion disrupts a brain area known as the Thalamus, compromising its ability to communicate with other brain areas. Pixabay

Researchers have discovered how a Genetic Alteration that increases the risk of developing Autism and Tourette’s impairs brain communication.

People with a genetic deletion known as chromosome 2p16.3 deletion often experience developmental delay and have learning difficulties.

They are also around 15 times more likely to develop Autism and 20 times more likely to develop Tourette’s Syndrome, but the mechanisms involved are not completely understood.

Using brain imaging studies, neuroscientists showed that deletion of the gene impacted by 2p16.3 deletion (Neurexin1) have impacts on the function of brain regions involved in both conditions.

This genetic deletion disrupts a brain area known as the Thalamus, compromising its ability to communicate with other brain areas, said the study published in the journal Cerebral Cortex.

“We currently have a very poor understanding of how the 2p16.3 deletion dramatically increases the risk of developing these disorders,” said lead researcher Neil Dawson of Lancaster University in Britain.

“However, we know that the 2p16.3 deletion involves deletion of the Neurexin1 gene, a gene that makes a protein responsible for allowing neurons to communicate effectively,” Dawson said.

Deletion of the Neurexin1 gene affects brain areas involved in Autism and Tourette’s including the Thalamus, a collection of brain regions that play a key role in helping other brain areas communicate with each other.

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Researchers have discovered how a Genetic Alteration that increases the risk of developing Autism and Tourette’s impairs brain communication. Pixabay

Changes were also found in brain regions involved in processing sensory information and in learning and memory.

Importantly, the researchers also found that the ability of the Thalamic brain regions to communicate with other brain areas was impaired by the genetic deletion.

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They then tested the ability of a low dose of the drug Ketamine, which is used clinically at higher doses as an anesthetic, to normalise the alterations in brain function induced by genetic deletion.

“Intriguingly our data suggest that Ketamine can restore some aspects of the brain dysfunction that results from 2p16.3 deletion and suggests that ketamine, or other related drugs, may be useful in treating some of the symptoms seen in autism and Tourette’s,” Dawson said. (IANS)